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PTSD and Neurophysiology [Vol 1/Num 3]By: Gary Maguire, M.Sc., P.T. http://www.linkedin.com/pub/gary-maguire-m-sc-p-t/7/455/449 PhysioSympath: The Path to Resilience © Welcome to Military Network’s new PTSD section. Articles that appear in this section are aimed at reducing the stigma of posttraumatic stress disorder (PTSD), strengthening resilience and providing new strategic solutions for PTSD treatment. Emerging neurophysiology and physiotherapy medical devices are being developed to support mental healthcare professionals. Your comments and feedback are welcome to improve the newsletter. MANAGING FIGHT OR FLIGHT SYMPTOMS High blood pressure (hypertension) and cardiovascular disease While the neuroendocrine system’s activation (a nerve cell that releases a chemical messenger, especially a neurohormone, directly into the bloodstream) during stress results in many cardiovascular effects that enhance performance in the act of fleeing or fighting the danger before us, such a response on a chronic basis leads to very pervasive and negative consequences (e.g., PTSD). One result of this activation is sympathetic innervations of the blood vessel walls, which while leading to greater blood flow to major muscles during a crisis, can lead to hypertension if sustained for a long-term. http://dx.doi.org/10.1001/jama.2009.1084 In addition other cardiac damage can occur when stress is not properly addressed and insufficient repair (i.e., down time) does not occur. While research in the 70’s focused on Type A personalities and the impact of always feeling hurried, research in the 90’s focused on the contribution of a specific element of Type A behavior – hostility – played in cardiovascular disease. The most recent research sheds light on the different role that each primary emotion associated with stress – anxiety, anger, and depression – uniquely contributes to the onset of recovery from hypertension, angina, myocardial infarction or other cardiovascular conditions. The emotional correlates of stress have been implicated as a primary cause of coronary artery disease. Two different mechanisms for such effects are the direct and indirect pathways through which anger/hostility, anxiety and depression lead to cardiovascular disease (CVD). Direct effects of stress on blood pressure and CVD outcomes
Indirect effects of stress on behaviors that lead to CVD
Interventions for minimizing the impact of stress on cardiovascular health
[AAA]= AWARENESS+ACT+ACCEPT
Posttraumatic Stress Disorder (PTSD) has real physiological consequences that cannot be ignored over time. The role of stress-related symptoms is vital for survival, yet sustained nature of such symptoms causes significant distress. Cortisol in the short term improves learning, memory, suppresses digestion, increases cardiovascular tone and increases glucose utilization. However, chronic activation leads to cardiomyopathy, digestive disorders and damage to parts of our brain (hippocampus) resulting in cognitive/emotional impairment. http://www.ptsd.va.gov/professional/pages/ptsd-physical-health.asp A good book to help better understand these relationships is: Why Zebras Don’t Get Ulcers, written by Robert M. Sapolsky a professor of biological sciences, neurology and neurological sciences at Stanford University. http://www.getabstract.com/summary/5656/why-zebras-dont-get-ulcers.html Posttraumatic stress disorder (PTSD) is a silent killer. Overcome the stigma and seek help from trained medical professionals. Your long term health depends on it. Act now! REAL WARRIORS GET REAL HELP: http://www.realwarriors.net/ Copyright © 2010 Gary Maguire, M.Sc., P.T., PhysioSympath©. All rights reserved. Related posts: |
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